Iga Nephropathy Treatment

IgA Nephropathy is the most common primary glomerulonephritis in the world. And about 20-40% patients progress to kidney failure. How to prevent it from progressing? The following 5 aspects will help you.

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5 Aspects Help Prevent IgA Nephropathy from Progressing to Kidney Failure

2018-10-14 03:33

5 Aspects Help Prevent IgA Nephropathy from Progressing to Kidney FailureIgA Nephropathy is the most common primary glomerulonephritis in the world. And about 20-40% patients progress to kidney failure. How to prevent it from progressing? The following 5 aspects will help you.

1. Immunosuppressants

Autoimmunity is involved in the pathological process of IgA nephropathy and progresses rapidly, so active treatment of immunosuppression is often required.

In general, steroid treatment should be used when urinary protein quantification is greater than or equal to 1g/ day, or when there is a pathological crescent.

If the condition is severe or progresses rapidly, steroid impact can be first applied before oral treatment.

2. To control blood pressure strictly

Keeping blood pressure stable not only inhibits glomerular damage but also significantly delays the progression of kidney disease.

The first choice is ACEI and ARBs, and the dose can be 2~4 times of the antihypertensive dose.

In addition, most patients need to take a variety of drugs to control blood pressure, often combined with drugs such as diuretics, long-term calcium antagonists, β-receptor blockers and so on.

3. To lower blood lipid

Patients with IgA nephropathy generally have increased blood cholesterol levels and can take statin lipid-lowering drugs.

4. To eliminate edema

Thiazide diuretics or loops diuretics remove excess fluid from the body and help control blood pressure.

5. Anti-infection

If there are also bacterial infections, antibiotics can be used to fight infection.

Then can IgA Nephropathy be cured? This is a concern for many patients.

In the opinion of doctors, to control blood pressure below 130/80 mmHg and urine protein quantification below 0.3g /d is clinical cure.

While in the opinion of patients, they think that “all indexes become normal” is cure.

But actually, already sclerotic glomeruli cannot be repaired, kidney failure that progresses gradually also is irreversible, and some symptom such as proteinuria, hematuria, and hypertension may exist for lifetime.

However, incomplete elimination of symptoms does not mean that there is no cure.

Is it not a cure to use drugs such as ACEI or ARBs to control symptoms at standard levels without cardiovascular and cerebrovascular complications and progression to renal failure?

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